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Nicholas Beazley-long

Employment

OrganisationRoleDepartmentStart dateEnd date
University of BristolKnowledge Exchange FellowNot specified2023-11-17
University of BristolPost DocNot specified2023-08-012023-11-16
University of NottinghamPost DocNot specified2019-09-012021-03-31
University of NottinghamPI & Research Fellow (Arthritis Research UK)Not specified2013-09-142019-09-14
University of BristolResearch Fellow (Skin Cancer Research Fund)Not specified2012-10-102013-09-01
King's College LondonResearch AssociateNot specified2012-04-012012-10-01
Boehringer Ingelheim Pharma GmbH and Co KG BiberachUndergraduate Trainee - placement yearNot specified2006-07-012007-07-01

Education

OrganisationDegree/titleDepartmentStart dateEnd date
University of BristolPhDNot specified2008-10-012012-07-01
University of ManchesterNeuroscience with Industry Placement YearNot specified2003-09-012008-07-01
University of ManchesterBiological Sciences Foundation YearNot specified2002-09-012003-07-01
Beechen Cliff School3 A levels, Maths, Physics and ComputingNot specified1998-09-012000-07-01

Works

TitleDOIPublished date
Diabetes-induced microvascular complications at the level of the spinal cord; a contributing factor in diabetic neuropathic pain.10.1113/jp2750672018-05
The physiological functions of central nervous system pericytes and a potential role in pain [version 1; referees: 2 approved]10.12688/f1000research.13548.12018-03-20
A role for pericytes in chronic pain?10.1097/spc.00000000000003422018-03
VEGFR2 promotes central endothelial activation and the spread of pain in inflammatory arthritis.10.1016/j.bbi.2018.03.0122018-03
Physiological role of vascular endothelial growth factors as homeostatic regulators10.1002/cphy.c1700152018
Sensory neuronal sensitisation occurs through HMGB-1-RAGE and TRPV1 in high-glucose conditions10.1242/jcs.2159392018
Alternative RNA splicing: contribution to pain and potential therapeutic strategy.10.1016/j.drudis.2016.06.0172016-11
Alternative RNA splicing: contribution to pain and potential therapeutic strategy- accepted for publicationNo DOI available.2016
Vascular endothelial growth factor-A165b prevents diabetic neuropathic pain and sensory neuronal degeneration.10.1042/CS201501242015-10
Novel mechanisms of resistance to vemurafenib in melanoma - V600E B-Raf reversion and switching VEGF-A splice isoform expressionNo DOI available.2015
Novel mechanisms of resistance to vemurafenib in melanoma - V600E B-Raf reversion and switching VEGF-A splice isoform expression.No DOI available.2015
Regulation of alternative VEGF-A mRNA splicing is a therapeutic target for analgesia.10.1016/j.nbd.2014.08.0122014-11
VEGF-A165b is an endogenous neuroprotective splice isoform of vascular endothelial growth factor A in vivo and in vitro.10.1016/j.ajpath.2013.05.0312013-09
VEGF-A165b is cytoprotective and antiangiogenic in the retina.10.1167/iovs.09-42962010-08

Octopus publications